ISSN: 2044-9038 (Print) 2044-9046 (Electronic)

Clinical Practice

Pharmacology And Clinical Toxicology Journals

Journal of Pharmacology & Clinical Toxicology dedicated to publish full-length papers, critical reviews, mini-reviews and short communications altogether fields of toxicology, basic and clinical pharmacology. This includes experimental animal pharmacology and toxicology, biochemical and cellular pharmacology and toxicology. It also includes all aspects of clinical pharmacology: pharmacokinetics, pharmacodynamics, therapeutic drug monitoring, drug interactions, pharmacogenetics, pharmacoepidemiology, pharmacovigilance, pharmacoeconomics, randomized controlled clinical trials and rational pharmacotherapy. As a neighborhood JSciMed Central Open Access platform, Journal of Pharmacology & Clinical Toxicology supports the scientific innovation and advancement in clinical pharmacology: pharmacokinetics, pharmacodynamics, therapeutic drug monitoring, drug interactions, pharmacogenetics, pharmacoepidemiology, pharmacovigilance, pharmacoeconomics research community by increasing access to see reviewed Open Accessscientific literature. JSciMed Central also brings multiple internationally peer reviewed, Open Access Journals under one roof thereby encouraging knowledge sharing, collaboration and promotion of interdisciplinary science.Cells become cancerous after being faced with several cellular insults, including DNA damages caused by oxidative stresses. Mitochondria are the main endogenous source of reactive oxygen species (ROS) because most ROS are generated by the oxidative phosphorylation processes that occur in mitochondria. ROS induce oxidative lesions and base alkylation in mtDNA that are thought to be repaired via BER pathway. However, the DNA repair proteins and other components/factors of BER pathway are not produced in the mitochondria but are translocated from cytoplasm or nucleus.  

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